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选择性抑制活化T淋巴细胞的功能—从中药及其成分中发现的崭新作用模式 被引量:1

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作者 徐强
出处 《哈尔滨商业大学学报(自然科学版)》 CAS 2001年第2期7-9,共3页 Journal of Harbin University of Commerce:Natural Sciences Edition
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  • 1徐强,王蓉,徐丽华.迟发型变态反应机制致肝损伤的动物模型的建立[J].中国免疫学杂志,1993,9(5):287-290. 被引量:9
  • 2[2]Xu Q, Wang R, Jiang J. et al. Liver injury model induced in mice by a cellular immunologic mechanism-Delayed-type hypersensitivityinduced liver injury to picryl chloride and phenotype of effector cell.mmunol, 1996, 167: 38-43.
  • 3[3]Xu Q, Jiang J, Cao J, et al. LFA-1/ICAM-1 interaction is essentially involoved in the pathogenesis of delayed-type hypersensitivity-induced liver injury to picryl chloride. Life Sciences 1998, 62:1281-1292.
  • 4[4]Xu Q, CaoJ, Wu F, et al. Role of Th1/Th2 cytokines in regulating the liver injury induced by delayed type hypersensitivity to picryl chloride. Liver 1999, in press.
  • 5[6]Chen X, Cao J and Xu Q. Endotoxin exacerbates the immunological liver injury in the cooperation of intefferon-γ. Submitted to J Hepatol.
  • 6[7]Xu Q. Wu F, Zhang B, et al. One-shot delayed-type hypersensitivity reaction in the mouse liver causes a sustained liver injury to picryl chloride. Life Sciences 1997; 60: 2417-2425.
  • 7[8]Xu Q, Wu F, Jiang J, et al. role of CD4+ and CD8+ T cells in regulating the chronic development of liver injury induced by delayed-type hypersensitivity to picryl chloride. Int Archs Allergy Immun.
  • 8[9]Xu Q, Lu J, Wang R, et al. Liver injury model induced in mice by a cellular immunologic mechanism-Study for use in immunopharmacological evaluations. Pharmacol Res, 1997, 35: 273-278.
  • 9徐强,赵红,杭秉茜.蒺藜对迟发型变态反应的抑制作用[J].中国药科大学学报,1991,22(1):12-16. 被引量:19
  • 10徐强,徐丽华.龙胆对迟发型变态反应的抑制作用[J].中药药理与临床,1993,9(5):25-26. 被引量:9

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