摘要
Objective To study the cavernosa hemodynamics in diabetic erectile dysfunction ( ED).Methods 22 diabetic and 35 psychic ED patients were studied by intracavernosum injection of a mixture papaver-ine and phentolamine (30/1mg) to assess the hemodynamics changes of the corpus cavernosum by means of colour duplex ultrasonography. Results The average hemodynamics data of the diabetic ED patients vs that of the psy-chogenic ED patients in terms of peak flow velocity (PFV):20. 06±7.15cm/s vs 35. 82±9. 41cm/s, end diastolic velocity (EDV) : 8. 82±0. 35cm/s vs 5. 51±0. 42cm/s,artery diameter (Ad) : 0. 78±0. 25cm vs 1. 01±0. 42cm, vein diameter ( Vd) : 1.05±0. 32mm vs 1.21±0. 45mm, resistance index(RI) : 0. 72±0. 28 vs 0. 98±0. 31,mean velocity of artery (MV):6. 71±0. 27cm/s vs 10. 31±3. 32cm/s, dorsal deep vein flow(DDVF) : 28. 81±6. 32cm/ s vs 25. 74±0. 58cm/s. Stasticstical differences existed in PFV, Ad,RI and MV(P <0. 01). The arterial wall is thick and rigid in diabetic ED patients. Conclusion Atheroscleorsis and veno-occlusive dysfunction of the corpus cavernosum are essential to the development of diabetic ED.
To study the cavernosa hemodynamics in diabetic erectile dysfunction (ED).Methods 22 diabetic and 35 psychic ED patients were studied by intracavernosum injection of a mixture papaverine and phentolamine ( 30/ l mg ) to assess the hemodynamics changes of the corpus cavernosum by means of colour duplex ultrasonography. Results The average hemodynamics data of the diabetic ED patients vs that of the psychogenic ED patients in terms of peak flow velocity ( PFV) : 20. 06 + 7. 15cm/s vs 35. 82 +9.41cmfs, end diastolic velocity (EDV) : 8. 82 + 0. 35cmf s vs 5.51 + 0. 42cm/s, artery diameter (Ad) : 0. 78 + 0. 25cm vs 1. 01 + 0. 42cm,vein diameter (Vd) : 1.05 +0. 32mm vs 1.21 +0.45mm, resistance index(R1) : 0. 72 +0. 28 vs 0. 98 +0. 31 ,mean velocity of artery ( MV) :6. 71 +0. 27cm/s vs 10. 31 +3.32cmfs, dorsal deep vein flow( DDVF) ： 28. 81 +6. 32cm!s vs 25. 74 + 0. 58cm/s. Stasticstical differences existed in PFV, Ad,R1 and MV( P <0.01 ). The arterial wall is thick and rigid in diabetic ED patients. Conclusion Atheroscleorsis and veno-occlusive dysfunction of the corpus cavernosum are essential to the development of diabetic ED.
