摘要
通过实验研究得出以下结论:(1)一次急性运动可造成红细胞的破坏增多,但小负荷的运动训练可以通过增强机体的抗氧化能力,改善红细胞膜性能等途径,最终增加动物血液中红细胞的数量,使其有氧代谢能力及抵抗力竭运动造成的能力均提高。大负荷的训练结果不但使红细胞的破坏增加,且单个红细胞的机能也明显下降,在训练结束后可能发生运动性贫血。(2)大负荷训练后RBCM流动性下降,与其丙二醛(MDA)含量及抗氧化酶SOD活性的变化高度相关。
The study investigated the training effect upon different exercise functions under different work loads. The following conclusions were drawn: (1)Acute exercise caused more the destruction of RBC but light load training ultimately improved the amount of RBC in the blood by enhancing the body's capability for anti-oxidation thus improving the function of RBCM. As a result, both the aerobic capacity and the ability to limit damage caused by exhaustive exercise were improved. Heavy load training lead to not only more RBC destruction but also a notable decline in function of individual RBC in the exercising animal by the end of training. (2)Powerful deforming ability in RBC made the carriage of oxygen more efficient. By the end of heavy load training, RBCM fluidity decline was highly related to the changes of MDA content and SOD activity on RBCM which indicated that training under different loads caused different reaction within the body lead to a discrepancy in exercising level.
出处
《天津体育学院学报》
CAS
CSSCI
北大核心
2004年第4期56-59,共4页
Journal of Tianjin University of Sport
