1Ji C, Kaplowitz N. Betaine decreases hyperhomocysteinemia, endoplasmic reticulum stress, and liver injury in alcohol-fed mice. Gastroenterology,2003,124(5): 1488-1499.
2Foo NC, Ahn BY, Ma X, et al. Celluler vacuolization and apoptosis induced by HBV large surface protein. Hepatology, 2002, 36(6): 1400-1407.
3Rodrigues CM, Ma X, Linehan Stieer C, et al. Ursodeoxycholic acid prevents cytochrome c release in apoptosis by inhibiting mitochondrial membrane depolarization and channel formation. Cell Death Differ, 1999, 6(9): 842-854.
4Pavio N, Romano PR, Graczyk TM, et al. Protein synthesis and endoplasmic reticulum stress can be modulated by the hepatitis C virus envelope protein E2 through the eukaryotic initiation factor 2alpha kinase PERK.J Virol,2003,77(6): 3578-3585.
5Tardif KD, Mori K, Siddiqui A. Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway. J Virol, 2002,76(15): 7453-7459.
6FerriKF, Kroemer G. Organelle-specific initiation of cell death pathways.Nat Cell Biol,2001,31 ( 11 ): E255-263.
7Mori K. Tripartite management of unfolded proteins in the endoplasmic reticulum. Cell, 2000,101 (5): 451-454.
8Wei MC,Zong WX,Cheng EH. Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death. Science, 2001, 292(5517) :727-730.
9Xie Q, Khaoustov VI, Chung CC, et al. Effect of tauroursodeoxycholic acid on ER stress -induced caspase-12 activation. Hepatology, 2002, 36(3): 592-601.
10Rizzuto R,Pinton P, Carrington, W, et al. Close contacts with the endoplasmic reticulum as determinants of mitochondrial Ca2+ responses,et al.Science, 1998,280(5370): 1763-1766.
