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颈动脉粥样硬化兔模型的建立 被引量:5

Establishment of the carotid atherosclerosis model in rabbits
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作者 赵瑾 迟路湘
出处 《中国脑血管病杂志》 CAS 2005年第11期522-524,共3页 Chinese Journal of Cerebrovascular Diseases
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  • 2[2]Kullo IJ, Gau GT, Tajik AJ. Novel risk factors for atherosclerosis. Mayo Clin Proc, 2000, 75: 369-380.
  • 3[3]Ghazalpour A, Doss S, Yang X, et al. Thematic review series: the pathogenesis of atherosclerosis. Toward a biological network for atherosclerosis. J Lipid Res, 2004,45: 1793-1805.
  • 4[4]Fan J, Watanabe . Transgenic rabbits expressing human apolipoprotein (a) . J Atheroscler Thromb, 2000, 7: 8-13.
  • 5[5]Wilson RB, Miller RA, Middleton CC, et al. Atherosclerosis in the rabbits fed a low cholesterol diet for five years.Arteriosclerosis, 1982, 2:228-241.
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  • 7[7]Nordestgaard BG, Wootton R, Lewis B. Selective retention of VLDL, IDL, and LDL in the arterial intima of genetically hyperlipidemic rabbits in vivo. Molecular size as a determinant of fractional loss from the intima-inner media. Arterioscler Thromb Vasc Biol, 1995, 15: 534-542.
  • 8[8]Brousseau ME, Hoeg JE. Transgenic rabbits as models for atherosclerosis research. Lipid Res, 1999, 40: 365-375.
  • 9[9]Bosze Z, Hiripi L, Carnwath JW, et al. The transgenic rabbit as model for human diseases and as a source of biologically active recombinant proteins. Transgenic Res,2003, 12: 541-553.
  • 10[10]Fan J, Shimoyamada H, Sun H, et al. Transgenic rabbits expressing human apolipoprotein (a) develop more extensive atherosclerotic lesions in response to a cholesterol-rich diet. Arterioscler Thromb Vasc Biol, 2001, 21: 88-94.

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