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细胞凋亡与高氧性肺损伤 被引量:8

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出处 《中国新生儿科杂志》 CAS 2006年第5期314-316,共3页 Chinese Journal of Neonatology
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参考文献21

  • 1Riccio VD,Turl MV,Post M.Apoptosis in lung development and neonatal lung injury.Pediatr Res,2003,55:1-7.
  • 2Schittny JC,Djonov V,Fine A,et al.Programmed cell death contributes to postnatal lung development.Am J Respir Cell Mol Biol,1998,18:786-793.
  • 3Hargital B,Szabo V,Hajdu J,et al.Apoptosis in various organs of preterm infants:histopathologic study of lung,kidney,liver,and brain of ventilated infants.Pediatr Res,2001,50:110-114.
  • 4Barazzone C,Horowitz S,Donati YR,et al.Oxygen Toxicity in mouse lung:pathways to cell death.Am J Respir Cell Mol Biol,1998,19:573-581.
  • 5McGrath-Morrow SA,Stahl J.Apoptosis in neonatal murine lung exposed to hyperoxia.Am J Respir Cell Mol Biol,2001,25:150-155.
  • 6李玉祥,罗小平,廖玲洁,刘皖君,宁琴.高氧对新生大鼠肺caspase-3和p53基因表达及肺细胞凋亡的影响[J].中华儿科杂志,2005,43(8):585-590. 被引量:19
  • 7Auten RL,Whorton MH,Nicholas MS.Blocking neutrophil influx reduces DNA damage in hyperoxia-exposed newborn rat lung.Am J Respir Cell Mol Biol,2002,26:391-397.
  • 8Budinger GR,Tso M,McClintock DS,et al.Hyperoxia-induced apoptosis does not require mitochondrial reactive oxygen species and is regulated by Bcl-2 proteins.J Biol Chem,2002,277:15654-15660.
  • 9De Paepe ME,Mao Q,Chao Y,et al.Hyperoxia-induced apoptosis and Fas/FasL expression in lung epithelial cells.Am J Physiol Lung Cell Mol Physiol,2005,289:L647-L659.
  • 10Buccellato LJ,Tso M,Akinci OI,et al.Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cells.J Biol Chem,2004,279:6753-6760.

二级参考文献14

  • 1李玉祥,罗小平,廖玲洁,刘皖君,宁琴.高氧对新生大鼠肺细胞周期调节基因表达的影响[J].实用儿科临床杂志,2005,20(2):108-110. 被引量:5
  • 2Tokunaga K, Taniguchi H, Yoda K, et al. Nucleotide sequence of a full-length cDNA for mouse cytoskeletal β-aetin mRNA. Nucleic Acids Res, 1986, 14: 2829-2833.
  • 3Arai N, Nomura D, Yokota K, et al. lmmunologically distinct p53 molecules generated by alternative spicing. Mol Cell Biol, 1986, 6:3232 -3239.
  • 4Hargitai B, Szabo V, Hajdu J, et al. Apoptosis in various organs of preterm infants: histopathologic study of lung, kidney, liver, and brain of ventilated infants. Pediatr Res, 2001, 50: 110-114.
  • 5Lukkarinen HP, Laine J, Kaapa PO. Lung epithelial cells undergo apoptosis in neonatal respiratory distress syndrome. Pediatr Res,2003, 53: 254-259.
  • 6McGrath-Morrow SA, Stahl J. Apoptosis in neonatal murine lung exposed to hyperoxia. Am J Respir Cell Mol Biol, 2001, 25: 150-155.
  • 7Constance Barazzone, Stuart Homwitz, Yves R, et al. Oxygen Toxicity in Mouse Lung: Pathways to Cell Death. Am J Respir Cell Mol Biol, 1998, 19: 573-581.
  • 8Sharon A, McGrath-Morrow, Jennifer Stahl. Growth arrest in A549 cells during hyperoxic stress is associated with decreased cyclin B1 and increased p21 ( Wafl/Cipl/Sdil ) levels. Biochim Biophys Acta, 2001,1538 : 90-97.
  • 9Schittny JC, Djonov V, Fine A, et al. Programmed cell death contributes to postnatal lung development. Am J Respir Cell Mol Biol, 1998,18 : 786-793.
  • 10Bardales RH, Xie SS, Schaefer RF, et al. Apoptosis is a majorpathway responsible for the resolution of type Ⅱ pneumocytes in acute lung injury. Am J Pathol, 1996, 149 : 845-852.

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二级引证文献52

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