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评估肥厚型心肌病的危险因素 被引量：2

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摘要 肥厚型心肌病(hypertrophic cardiomyopathy,HCM)通常表现为:特殊的心电图表现,收缩期心室造影时异常的左室图形,心脏超声显示典型的不对称性增生以及舒张期左室异常充盈.自从大约五十多年前这些肥厚型心肌病特殊的表现被描述出来以后,HCM一直受到临床医生和研究人员的关注.关于这种疾病的某些方面引起了广泛争论,例如某些病人的室内血流压力梯度的形成机制(和存在性)问题.
作者 赵超
出处 《中国分子心脏病学杂志》 CAS 2003年第4期249-250,共2页 Molecular Cardiology of China
关键词 肥厚型心肌病 危险因素 评估 心电图表现 心室造影 超声显示 研究人员 临床医生
分类号 R542.2 [医药卫生—心血管疾病]
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同被引文献30

  • 1刘健,何作云,王培勇.细胞核CaMK和Calcineurin参与大鼠心肌肥厚的发生[J].中国分子心脏病学杂志,2002,2(1):10-13. 被引量：1
  • 2[1]Maron BJ,Gardin JM,Flack JM,et al.Prevalence of hypertrophic cardiomyopathy in a general population of young adults.Echocardiographic analysis of 4111 subjects in the CARDIA Study.Coronary Artery Risk Development in (Young) Adults.Circulation,1995,92(4):785-789.
  • 3[2]Zou Y,Song L,Wang Z,et al.Prevalence of idiopathic hypertrophic cardiomyopathy in China:a population-based echocardiographic analysis of 8080 adults.Am J Med,2004,116(1):14-18.
  • 4[4]Hardie DG.AMP-activated protein kinase:the guardian of cardiac energy status.J Clin Invest,2004,114 (4):465-468.
  • 5[5]Russell RR,Bergeron R,Shulman Gl,et al.Translocation of myocardial GLUT4 and increased glucose uptake through activation of AMPK by AICAR.Am J Physiol,1999,277(2 Pt 2):H643-649.
  • 6[6]Luiken JJ,Coort SL,Willems J,et al.Contraction-induced fatty acid translocase/CD36 translocation in rat cardiac myocytes is mediated through AMP-activated protein kinase signaling.Diabetes,2003,52(7):1627-1634.
  • 7[7]Kudo N,Barr A J,Barr RL,et al.High rates of fatty acid oxidation during reperfusion of ischemic hearts are associated with a decrease in malonyl-CoA levels due to an increase in 5'-AMP-activated protein kinase inhibition of acetyl-CoA carboxylase.J Biol Chem,1995,270 (29):17513-17520.
  • 8[8]Lowey S.Functional consequences of mutations in the myosin heavy chain at sites implicated in familial hypertrophic cardiomyopathy.Trends Cardiovasc Med,2002,12(8):348-354.
  • 9[9]Sweeney HL,Feng HS,Yang Z,et al.Functional analyses of troponin T mutations that cause hypertrophic cardiomyopathy:insights into disease pathogenesis and troponin function.Proc Natl Acad Sci U SA,1998,95(24):14406-14410.
  • 10[10]Michele DE,Albayya FP,Metzger JM.Direct,convergent hypersensitivity of calcium-activated force generation produced by hypertrophic cardiomyopathy mutant alpha-tropomyosins in adult cardiac myocytes.Nat Med,1999,5(12):1413-1417.

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中国分子心脏病学杂志
2003年 第4期

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