2Kanter D, Lindheimer MD, Wang E, et al. Angiogenic dys- function in molar pregnancy [J]. Am J Obstet Gynecol, 2010, 202 (2): 184.
3Levine RJ, Maynard SE, Qian C, et al. Circulating angiogenic factors and the risk of preeclampsia [J]. N Engl J Med, 2004, 350 (7) 672-683.
4Venkatesha S, Toporsian M, Lam C, et al. Soluble endoglin contributes to the pathogenesis o preeclampsia [J]. Nat Med,2006, 12 (6) 642-649.
5Roh CR, Budhraja V, Kim HS, et al. Microarray-based iden- tification of differentially expressed genes in hypoxic term hu- man trophoblasts and in placental villi of pregnancies with growth restricted fetuses [ J]. Placenta, 2005, 26 ( 4 ) 319-328.
6Okamoto A, Endo H, Kalionis B, et al. IGFBP1 and Fol- listatin-like 3 genes are significantly up-regulated in expression profiles of the IUGR placenta [J]. Placenta, 2006, 27 (2-- 3) .. 317-321.
7McCarthy C, Cotter FE, McElwaine S, et al. Altered gene expression patterns in intrauterine growth restriction: potential role of hypoxia [J]. Am J Obstet Gynecol, 2007, 196 (i): 70.
8Struwe E, Berzl G, Schild R, et al. Microarray analysis o{ placental tissue in intrauterine growth restriction [J]. Clin En- docrinol, 2010, 72 (2): 241-247.
9Wyatt SM, Kraus FT, Roh CR, et al. The correlation be- tween sampling site and gene expression in the term human pla- centa [J]. Placenta, 2005, 26 (5): 372-379.
10Tzschoppe AA, Struwe E, D rr HG, et al. Differences in gene expression dependent on sampling site in placental tissue of fetuses with intrauterine growth restriction [J]. Placenta, 2010, 31 (3) 178-185.