2Cooperative Study Group of Phase Ⅲ Clinical Trial on Meisoindico,Tianjin 300020.甲异靛治疗慢性粒细胞白血病 Ⅲ 期临床观察[J].中华血液学杂志,1997,18(2):69-72. 被引量：16
3Nucifora G,Rowley JD.AML1 and the 8 ;21 and 3 ;21 translocations in acute and chronic myeloid leukemia.Blood,1995,86:1-14.
4Johansson B,Fioretos T,Mitelman F.Cytogenetic and molecular genetic evolution of chronic myeloid leukemia.Acta Haematol,2002,107:76-94.
5Rubin CM,Larson RA,Bitter MA,et al.Association of a chromosomal 3 ;21 translocation with the blast phase of chronic myelogenous eukemia.Blood,1987,70:1338-1342.
6Nucifora G,Begy CR,Erickson P,et al.The 3;21 translocation in myelodysplasia results in a fusion transcript between the AML1 gene and the gene for EAP,a highly conserved protein associated with the Epstein-Barr virus small RNA EBER 1.Proc Natl Acad Sci U S A,1993,90:7784-7788.
7Mitani K,Ogawa S,Tanaka T,et al.Generation of the AML1-EVI-1 fusion gene in the t(3 ;21) (q26; q22) causes blastic crisis in chronic myelocytic leukemia.EMRO J,1994,13:504-510.
8Nucifora G,Begy CR,Kobayashi H,et al.Consistent intergenic splicing and production of multiple transcripts between AML1 at21q22 and unrelated genes at 3q26 in (3 ;21)(q26 ;q22) translocations.Proc Natl Acad Sci U S A,1994,91:4004-4008.
9Sacchi N,Nisson PE,Watkins PC,et al.AML1 fusion transcripts in t(3 ;21) positive leukemia:evidence of molecular heterogeneity and usage of splicing sites frequently involved in the generation of normal AML1 transcripts.Genes Chromosomes Cancer,1994,11:226-236.
10Lahortiga I,Vazquez I,Agirre X,et al.Molecular heterogeneity in AML/MDS patients with 3q21q26 rearrangements.Genes Chromosomes Cancer,2004,40:179-189.
