期刊文献+

载脂蛋白E基因敲除小鼠钟基因、凋亡相关基因及动脉粥样硬化相关基因表达节律的改变 被引量:2

下载PDF
导出
出处 《中国动脉硬化杂志》 CAS CSCD 2007年第7期545-545,共1页 Chinese Journal of Arteriosclerosis
  • 相关文献

同被引文献24

  • 1Yannielli PC, Molyneux PC, Harrington ME, et al. Ghrelin effects on the circadian system of mice [ J ]. Journal of Neuro- science, 2007(27( 1 ) :2890 -2895.
  • 2Turek FW, Joshu C, Kohsaka A, et al. Obesity and metabolic syndrome in circadian clock mutant mice [ J]. Science, 2005, 308 (5724) : 1043 - 1045.
  • 3Zhang X,Zhao F,Xu Ch. Circadian rhythm disorder of thrombosis and thrombolysis -related gene expression in apolipoprotein E knock -out mice[J]. Int J Mol Med,2008,22(2) :149 -153.
  • 4Schiopu A, Frend us B, Jansson B, et al. Recombinant antibod- ies to an oxidized low - density lipoprotein epitope induce rapid regression of atherosclerosis in apobec - 1 ( - / - )/low - densi- ty lipoproteinreceptor ( -/- ) mice [ J 1. Am Coil Cardiol, 2007,50(24) :2313 -2318.
  • 5Bonomini F, Tengattini S, Fabiano A, et al. Atherosclerosis and oxidative stress [ J ]. Histol Histopathol, 2008,23 ( 3 ) : 381 - 390.
  • 6Aikawa E, Nahrendorf M, Figueiredo JL, et al. Osteogenesis as- sociates with inflammation in early - stage atherosclerosisevalu- ated by molecular imaging in vivo [ J ]. Circulation, 2007,116 (24) :2841 -2850.
  • 7Van Wanrooij EJ, de Jager SC, van Es T. CXCR3 antagonist NBI- 74330 attenuates atherosclerotic plaque formation in LDL receptor - deficient mice [ J ]. Arterioscler Thmmb Vasc Biol, 2008,28 (2) :251 - 257.
  • 8Balsalobre A. Clock genes in mammalian peripheral tissues[ J]. Cell Tissue Res ,2002,309 ( 1 ) : 193 - 199.
  • 9Durgan DJ, Hotze MA, Tomlin TM, et al. The intrinsic circa- dian clock within the cardiomyocyte [ J ]. Am J Physiol heart circ Physiol,2005, 289 :H153 - H1541.
  • 10Young ME. The circadian clock within the heart: potential in- fluence on myocardial gene expression, metabolism and function AJP [J]. Heart, 2006, 290:1-16.

引证文献2

二级引证文献9

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部