摘要
硫化氢可以在哺乳动物循环系统内源性产生。硫化氢能减轻心肌缺血／再灌注损伤，这一保护作用的机制包括：清除自由基、抑制炎症反应、保护线粒体功能、有关信号通路激活、ATP敏感性钾通道开放。
Hydrogen sulfide （H2S）, produced endogenously in mammalian cardiovascular system, attenuates ischemia/reperfusion-induced myocardial injury. The protective mechanism involves in free radical scavenging, inflammation inhibition, mitochondria preservation, signal pathway activation and ATP-sensitive potassium channel opening.
出处
《国际麻醉学与复苏杂志》
CAS
2008年第4期310-314,共5页
International Journal of Anesthesiology and Resuscitation
关键词
硫化氢
心肌保护
缺血/再灌注损伤
hydrogen sulfide
myocardial protection
ischemia/reperfusion injury
