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药物对心肌梗死后心脏胶原重构干预的研究进展 被引量:5

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摘要 心力衰竭(简称心衰)是由于初始心肌损伤(如心肌梗死、心肌病、血流动力学负荷过重、炎症等)引起心肌结构和功能的变化,最后导致心室泵血和(或)充盈功能低下。目前己明确,导致心力衰竭发生发展的基本机制是心肌重构。心肌重构是一系列复杂的分子和细胞机制引起的心肌细胞结构、功能及表型的改变,临床上表现为心肌肌重和心室容量的增加,以及心室形状的改变,横径增加呈球状。
作者 刘相飞 吴强
出处 《中国心血管病研究》 CAS 2008年第9期706-708,共3页 Chinese Journal of Cardiovascular Research
基金 贵州省科学技术基金资助项目[黔基合计字(2003)3043] 贵州省高层次人才科研条件特助经费项目[黔人领函(2005)14]
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共引文献3770

同被引文献28

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