4[1]Ross R. Atherosclercsis: an inflammatory disease.disease. N Engl J Med, 1999,340:115-126
5[2]PasceriV, Willerson JT, Yeh ETH. Direct proinflammatory effect of C-reactive protein on human endothelial cells. Circulation, 2000, 102:2 165-168
6[3]Pasceri V, Chang J, Willerson JT, etal. Modulation of C-reactiveprotein-mediated monocyte chemoattractant protein-1 induction in human endothelial cells by anti-atherosclemsis drugs. Circulation, 2001. 103:2 531-534
7[4]Zwaka TP, Hombach V, Torzewski J. C-reactive protein-mediated low density lipoprotein uptake by macrophages implications for atherosclerosis. Circulation,2001, 103: 1194-197
8[5]Bhakdi S, Torzewski M, Klouche M, et al. Complement and athemgenesis:binding of CRP to degraded, nonoxidized LDL enhances complement activation.Arterisocler Thromb Vasc Biol, 1999, 19:2 348-354
9[6]Griselli BM, Herbert J, Hutchirnson WL, et al. C-eractive protein and complement are important mediators of tissue damage in acute myocardial infarction. J Exp Med, 1999, 190: 1733-739
10[7]Zhang YX, Cliff WJ, Schoefl GI, et al. Coronary C-reactive protein distribution: its relation to development of atherosclerosis. Atherosclerosis, 1999, 145:375-379
8Roberts WL, Sedrick R, Moulton L, et al. Evaluation of four automated high-sensitivity C-reactive protein meth- ods: implications for clinical and epidemiological applica- tions[J]. Clin Chem,2000,46(4) :461-468.
9Daraid J, Steven EW, Marc GJ, et al. Growth hormone at- tenuates the acute-phase response to thermal injury[J]. Arch Surg, 1997,132(11) : 1171-1175.
