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慢性肝病金属蛋白酶组织抑制因子-1基因表达的研究 被引量:5

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摘要 慢性肝炎、肝硬化病理基础是有过多的胶原纤维在肝脏中沉积。其发生与胶原蛋白的合成代谢及降解代谢有关。参与胶原蛋白降解代谢的是基质金属蛋白酶类(matrixmetaloproteinases,MMPs);它们的生物活性受其特异性抑制物———金属蛋白酶组织...
出处 《中华消化杂志》 CAS CSCD 北大核心 1999年第1期59-60,共2页 Chinese Journal of Digestion
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参考文献1

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同被引文献20

  • 1高峰,程留芳,王志强,杨云生.木苏丸对大鼠二甲基亚硝胺肝纤维化肝组织TIMP-1,MMP-2 mRNA表达的作用[J].世界华人消化杂志,2005,13(3):355-357. 被引量:4
  • 2马红,朱跃科,马雪梅,贾继东,王宝恩.γ-干扰素抑制肝纤维化大鼠Ⅰ、Ⅲ型胶原和基质金属蛋白酶组织抑制因子基因表达[J].肝脏,2005,10(2):92-94. 被引量:4
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