1Naka K,Muraguchi T,Hoshii T,et al.Regulation of reactive oxygen species and genomic stability in hematopoietic stem cells.Antioxidants Redox Signaling,2008,10:1883-1894.
2Kong Y,Zhou S,Kihm AJ,et al.Loss of alpha-hemoglobin stabilizing protein impairs erythropoiesis and exacerbates beta-thalassemia.J Clin Invest,2004,114:1457-1466.
3Chen ZX,Pervaiz S.Bcl-2 induces pro-oxidant state by engaging mitochondrial respiration in tumor cells.Cell Death Differ,2007,14:1617-1627.
6Jensen PD, Heickendorff L, Pedersen B, et al. The effect of iron chelation on haemopoiesis in MDS patients with transfusional iron overload. Br J Haematol, 1996 ; 94 (2) :288 - 299.
7Fibach E, Rachmilewitz EA. The role of antioxidants and iron chelators in the treatment of oxidative stress in thalassemia. Ann N Y Acad Sci, 2010; 1202:10-16.
8Prus E, Fibach E. Effect of iron chelators on labile iron and oxidative status of thalassaemic erythroid cells. Acta Haematol, 2010; 123(1) :14 -20.
9Zhao MF, Xie F, Li YM, et al. Increased intracellular concentration of reactive oxygen species mediated the deficient hematopoiesis of iron overload bone marrow. Blood, 2010; 116 (21) :4247a.
10Kruszewski M. Labile iron pool: the main determinant of cellular response to oxidative stress. Mutat Res, 2003 ; 531 ( 1 -2) :81 -92.