1Rao RV, Hermel E, Castro - Obregon S, etal. Coupling endoplasmic reticulum stress to the cell death progTame. Mechansim of caspase activation. J Biol Chem,2001,276 ( 36 ) :33869 - 33874.
2Kadowaki H, Nishitoh H, lchijo H. Survival and apoptosis signals in ER stress : the role of protein kinases [ J ]. J Chem Neuroanat,2004, 28(1 -2) :93 - 100.
3Robinson LC, Marchant JS. Enhanced Ca2 + leak from ER Ca2 + stores induced by hepatitis C NSSA protein[ J]. Biochem Biophys Res Commun ,2008,368 ( 3 ) :593 - 599.
4Pahl HL. Signal transduction from the endoplasmic reticulum to the cell nucleus Physiol Rev, 1999, (79) :683 - 701.
5Lai E, Teodoro T, Volchuk A. Endoplasmic Reticulum Stress : Sig- naling the Unfolded Protein Response. Physiology, 2007,22 ( 3 ) : 193 - 201.
10Shen J, Chen X, Hendershot L, etal. ER stress regulation of ATF6 localization by dissociation of BiP/GRP78 binding and unmasking of Golgi localization signals. Dev Ce11,2002,3:99 - 111.
4Benavides A, Pastor D, Santos P, et al. CHOP plays a pivotal role in the astrocyte death induced by oxygen and glucose deprivation. Glia. 2005,52(4) :261.
5Rutkowski DT, Arnold SM, Miller CN, et al. Adaptation to ER stress is mediated by differential stabilities of pro-survival and pro-apoptotic mRNAs and proteins. Plos Biol, 2006, 4( 11 ) :374.
6Petito CK, Pulsinelli WA. Delayed neuronal recovery and neuronal death in rat hippocampus following severe cerebral ischemia: possible relationship to abnormalities in neuronal processes. Cereb Blood Flow Metab,1984, 4(2) :194.
7Hu BR, Janelidze S, Ginsberg MD, et al. Protein aggregation after focal brain ischemia and reperfusion. Cereb Blood Flow Metab,2001, 21 (7) :865.
8Yoshida H, Haze K, Yanagi H, et al. Identification of the cis-acting endoplasmic reticulum stress response element responsible for transcriptional induction of mammalian glucose-regulated proteins. Involvemeut of basic leucine zipper transcription factors. J Biol Chem, 1998, 273(50) :33741.
9Paschen W, Gissel C, Linden T,et al. Activation of gadd153 expression through transient cerebral ischemia: evidence that ischemia causes endoplasmic reticulum dysfunction. Brain Res Mol Brain Res, 1998,60( 1 ) :115.
10Shibata M, Hattori H, Sasaki T, et al. Activation of caspase-12 by endoplasmic reticulum stress induced by transient middle cerebral artery occlusion in mice. Neuroscience, 2003, 118 (2) : 491.