期刊文献+

脑积水病理改变及发病机制研究现状 被引量:4

原文传递
导出
摘要 脑积水是神经外科常见疾病,可以单纯发生或合并于其他疾病。脑积水由不同病因导致,主要有外伤、脑室内出血、肿瘤和感染。传统观点认为脑积水的病理机制主要分为3种,即脉络丛产生脑脊液过多、脑脊液吸收障碍和脑脊液循环受阻。不论什么原因导致的脑积水,发病机制和病理改变均十分相似,其相关研究随着实验动物模型的广泛应用而逐渐加深,但是仍没有定论。
作者 徐昊 王占祥
出处 《中华神经外科杂志》 CSCD 北大核心 2012年第9期969-970,共2页 Chinese Journal of Neurosurgery
基金 中国博士后基金资助项目(20080440728) 福建省自然科学基金面上项目(2009D002)
  • 相关文献

参考文献23

  • 1孟辉,张礼均,邓聪颖,王俊伟,谢宾,陈杰,冯华.猪实验性脑室出血后慢性脑积水模型的建立及蛛网膜下腔脑脊液循环的改变[J].中华神经外科杂志,2007,23(8):625-628. 被引量:12
  • 2陈东汉,王占祥.脑积水动物模型研究进展[J].中华实验外科杂志,2010,27(10):1557-1558. 被引量:2
  • 3Yang J, Dombrowski SM, Deshpande A,et al. VEGF/VEGFR -2 changes in frontal cortex, choroid plexus, and CSF after chronic obstructive hydrocephalus. J Neurol Sci ,2010,296:39-46.
  • 4赵时雨,袁先厚,江普查,吴光耀,文志华.实验性脑积水幼鼠脑脊液中一氧化氮及谷氨酸浓度的变化[J].中国临床神经外科杂志,2009,14(10):601-602. 被引量:2
  • 5Chu SH, Feng DF, Ma YB, et al. Expression of' HGF and VEGF in the cerebral tissue of adult rats with chronic hydrocephalus after subarachnoid hemorrhage. Mol Med Report,2011,4:785-791.
  • 6Douglas MR,Daniel M ,Lagord C ,et al. High CSF transforming growth factor beta levels after subarachnoid haemorrhage: association with chronic communicating hydroeephalus. J Neurol Neurosurg Psychiatry, 2009,80:545 -550.
  • 7Brinar VV, Habek M, Ozreti6 D, et al. Isolated nontraunmtic abdu- cens nerve palsy. Acta Neurol Belg,2007,107 : 126-130.
  • 8Voelz K, Kondziella D, yon Rautenfeld DB, et al. A ferritin tracer study of compensatory spinal CSF outflow pathways in kaolin - induced hydrocephalus. Acta Neuropathol, 2007,113:569-575.
  • 9Cabuk B, Etus V, Bozkurt SU, et al. Neuroprotective effect of memantine on hippocampal neurons in infantile rat hydrocephalus. Turk Neurosurg, 2011,21:352-358.
  • 10Castej6n OJ. Submicroscopic pathology of human and experimental hydrocephalic cerebral cortex. Folia Neuropatho1,2010, 48:159-174.

二级参考文献60

共引文献23

同被引文献44

  • 1唐维国,何松彬,王和平,朱永礼,孙茂军,吴阿淼.CT灌注成像指导脑梗死超早期静脉溶栓治疗[J].浙江医学,2009(10). 被引量:4
  • 2王国平,詹荣举.重症颅脑损伤后脑积水的早期诊断及治疗[J].中华神经医学杂志,2007,6(1):80-81. 被引量:15
  • 3LP K. Hydrocephalus following severe traumatic brain injuryin adults. Incidence, timing, and clinical predictors duringrehabilitation. NeuroRehabilitation,2013,33(3): 473-480.
  • 4Zhang B , Li S. Cine - PC MR in assessment of cerebrospinalfluid velocity in the aqueduct of the midbrain correlated withintracranial pressure - Initial study. Medical Hypotheses,2012, (2) :227-230.
  • 5Bailey BM,Liesemer KM , Statler KD et al. Monitoring andprediction of intracranial hypertension in pediatric traumaticbrain injury : Clinical factors and initial head computed tomo-graphy. J Trauma Acute Care Surg ,2012 ,72 (1) :263-270.
  • 6Appelboom G,Piazza M , Zoller SD et al. Clinical trials indecompressive craniectomy after severe diffuse traumatic braininjury. World Neurosurgery , 2013, ( 5 ) : el 5 3-e 155 .
  • 7Woemle CM ’ Winkler KM , Burkhardt JK,et al Hydroceph-alus in 389 patients with aneurysm - associated subarachnoidhemorrhage. Clin Neurosci, 2013,20(6) : 824-826.
  • 8Gouello G. Hamel 0,Asehnoune K , et al. Study of thelong term results of decompressive craniectomy after severetraumatic brain injury based on a series of 60 consecutive ca-ses .Scientific World Journal, 2014, 310(1) :18.
  • 9De Bonis P,Mangiola A,Pompucci A , et al. DecompressiveCraniectomy and Hydrocephalus. Neurosurgery,2011, 68(6). E1777-E1778.
  • 10Linnemann M , Tibaek M,Kammersgaard LP. Hydrocephalusduring rehabilitation following severe TBI. Relation to recovery,outcome,and length of stay. Neurorehabilitation. 2014 , 35(4):755-761.

引证文献4

二级引证文献30

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部