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重症休克的发生机制 被引量:1

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作者 赵克森
出处 《中国病理生理杂志》 CAS CSCD 北大核心 2000年第10期938-939,共2页 Chinese Journal of Pathophysiology
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同被引文献33

  • 1赵克森.创伤性休克的新概念[J].中华创伤杂志,2005,21(1):29-31. 被引量:25
  • 2赵克森.重症难治性休克发生机制的若干进展[J].解放军医学杂志,2005,30(7):552-554. 被引量:26
  • 3Zhao KS. Hemorheologic events in severe shock[J]. Biorheology, 2005, 42(6): 463-477.
  • 4Zhao KS, Song R, Wang X. Mitochondrial dysfunction in severe hemorrhagic shock[J]. Advances in Medicine and Biology, 2014, 77: 125-133.
  • 5Song R, Bian H, Wang X, HuangX, ZhaoKS. Mitochondrialinjury underlies hyporeactivity of arterial smooth muscle in severe shock[J]. AmJHypertens, 2011, 24(1): 45-51.
  • 6Wang X, Song R, Chen Y, Zhao M, Zhao KS. Polydatin a new mitochondria protector for acute severe hemorrhagic shock treatment[J]. Expert Opin Investig Drugs, 2013, 22(2): 169-179.
  • 7Wang X, Song R, Bian HN, Brunk UT, Zhao M, Zhao KS. Polydatin, a natural polyphenol, protects arterial smooth muscle cells against mitochondrial dysfunction and lysosomal destabilization following hemorrhagic shock[J]. Am J Physiul Regul Integr Comp Physiol, 2012, 302(7): R805-Rg14.
  • 8ZhaoG, ZhaoY, PanB, LiuJ, HuangX, ZhangX, CaoC, Hou N, Wu C, Zhao KS, Cheng H. Hypersensitivity of BKCa to Ca2+ sparks underlies hyporeactivity of arterial smooth muscle in shock[J]. Circ Res, 2007, 101(5): 493-502.
  • 9Zhao KS, Huang X, Liu J, et al. New approach to treatment of shock. Shock. 2002, 18(2): 189-192.
  • 10Chart JK, Roth ], Oppenheim J], et M. Alarmins : awaiting a clinical response[J]. J Clin Invest, 2012, 122(8): 2711-2719.

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