1[1]Runyon BA. Historical aspects of treatment of patients with cirrhosis and ascites. Semin Liver Dis 1997; 17:163
2[2]Wensing G, Lotterer E,link I,et al. Urinary sodium balance in patients with cirrhosis: relationship to quantitative paramenters of liver function. Hepatology 1997; 26:1149
3[3]Moreau R, Lebrec D. Transduction of antinatriuretic signals in renal proximal tubular cells in cirrhosis: introduction to novel approaches to the treatment of sodium retention. J Hepatol 1998; 28:1064
4[4]Wong F. Sniderman K, BlendisL. The renal sympathetic and renin-angiotensin response to lower body negative pressure in well-compensated cirrhosis. Gastroenterology 1998; 115:397
5[5]Epstein M. A trial natriuretic factor and liver disease.In:Epstein M (ed.)The Kidney in Liver Disease. 4th ed.Philadelphia; Hanley & Belfus Inc. ; 1996:p339～358
6[6]Uemure M, Tsujii T, Kikuchi E, et al. Increased plasma levels of substance P and disturbed wanter excretion in patients with liver cirrhosis. Scand J Gastroenterol 1998;33: 860
7[7]Kanwar S, Kubes P,Tepperman B, et al. Nitric oxide synthase activity in portal-hypertensive and cirrhostic rats. J Hepatology 1996; 25: 85
8[8]Kojima H, Tsujimoto T, Uemura M,et al. Significance of increased plasma adrenomedullin concentration in patients with cirrhosis. J Hepatol 1998; 28: 840
9[9]Gutilini P. La Villa G. Marra F. et al. Pharmacokinetics and pharmacodynamics of torsemide and furosemide in patients with diuretic resistant ascites. J Hepatology 1996;25: 481
10[10]Gines P. Jimenez W. Aquaretic agents: a new potential treatment of dilutional hyponatremia in cirrhosis. J Hepato 1996; 24: 506
