摘要
在116只成功建立的大鼠脑缺血模型中，于缺血及再灌流各期对海马结构中DynA（1-13）、GABA及CCK的动态变化、缺血／再灌流神经损害的病理进行研究，发现脑缺血和再灌流期这三种物质的释放增多，且大剂量纳洛酮有防治DND的作用。
Cerebral ischemia/repercussion model of 112 rats was used to study the dynamic changes of DynA(1-13)、GABA、CCK and to observe the pathologic changes of brain ischemia during the ischemia/repercussion periods. The study demonstrated that the liberation of the above-mentioned 3 bioactive substances increased, and that the high dose of naloxone administration could prevent the pyramidal cells from DND (delayed neuronal death).
出处
《汕头大学医学院学报》
1997年第4期4-6,共3页
Journal of Shantou University Medical College
