期刊文献+

组织型激肽释放酶对大鼠急性脑缺血后GLUT-1表达的影响

下载PDF
导出
摘要 激肽释放酶-激肽系统(kallikre in-kinin system,KKS)在人体分布广泛,发挥重要生理的作用,尤其在血管再生方面发挥重要作用[1]。本研究通过观察组织型激肽释放酶(TK)对脑缺血后 GLUT-1蛋白表达的影响,
出处 《中国实验诊断学》 2014年第7期1052-1053,共2页 Chinese Journal of Laboratory Diagnosis
基金 吉林省科技厅社会发展计划项目(No.20090463)
  • 相关文献

参考文献11

  • 1Emam iN,Diam and isEP. Human tissue kallik reins: A road un-derconstrat ion[J]. Clin Chin Acta, 2007,381 (1) : 78.
  • 2Longa EZ, Weintein PR. Carson S, et al. Rerersible middle Cere bral aetery occlusion without crainietomy in rats [J]. Stroke, 1989,20(1) :84.
  • 3Xia CF, Yin H, Yao YY, et al. Kallikrein protects against ischemic stroke by inhibiting apoptosis and inflammation and promoting angiogenesis and neurogenesis [ J ]. Hum Gene Ther, 2006, 17 : 206.
  • 4Xia CF,Yin H,Borlongan CV,et al. Kallikrein gene transfer pro- tects afainst ischemic stroke by promoting glial cell migration and inhibiting apoptosis[J]. Hypertension, 2004,43 : 452.
  • 5Murakami H, Yayama K, Miao RQ, et al. Kallikrein gene delivery inhibits vascular smooth muscle cell growth and neointima forma- tion in the rat artery after balloon angioplasty[J]. Hypertension, 1999,34: 164.
  • 6倪耀辉,丁素菊.组织型激肽释放酶对脑缺血大鼠神经和血管的影响[J].中国神经精神疾病杂志,2009,35(3):173-175. 被引量:2
  • 7Nehlig A. Cerebral energy metabolism, glucose transport and blood flow:changes with maturation and adaplation tohypoglycae- mia[J]. Diabetes Metab, 1997,23(1) : 18.
  • 8Simpson IA,Appel NM, Hokari M, et al. Blood-brain barrier glu- cose transporter: effects of hypo-and hyperglycemia revisited[J]. J Neurochem,1999,72(1) :238.
  • 9Dobrogowska DH,Vorbrodt AW. Quantitative immunoeytochemical study of mouse brain[J]. J Histochem Cytochem, 1999,47 (8) : 1021.
  • 10Regina A, Morechoisne S, Borson ND, et al. Factors released by glucose deprived astrocytes enhance glucose transporter expres- sion and activity in rat brain endothelia cells[J]. Biochim Biophys Acta, 2001,1540(3) : 233.

二级参考文献16

  • 1Emami N, Diamandis EP. Human tissue kallikreins: A road under constration[ J]. Clin Chim Acta, 2007,381 (1) :78 -84.
  • 2Yousef GM, Kishi T, Diamandis EP. Role of kallikrein enzymes in the central nervous system[ J]. Clin Chim Acta, 2003, 329 ( 1 - 2) : 1 -8.
  • 3Longa EZ, Weinstein PR, Carlson L, et al. Reversible middle cerebral artery occlusion without craniectomy in rats [ J ]. Stroke, 1989, 20(1) : 84-91.
  • 4Chen J, Li Y, Wang L, et al. Therapeutic benefit of intravenous administration of bone marrow stromal cells after cerebral ischemia in rats[J]. Stroke, 2001, 32(4): 1005-1011.
  • 5Gerriets T, Li F, Silva MD, et al. The macrosphere model: evaluation of a new stroke model for permanent middle cerebral artery occlusion in rats[J]. J Neurosci Methods, 2003, 122(2) : 201 -211.
  • 6Lumenta DB, Plesnila N, Klasner B, et al. Neuroprotective effects of a postischemic treatment with a bradykinin B2 receptor antagonist in a rat model of temporary focal cerebral ischemia[J]. Brain Research, 2006,1069( 1 ) : 227 -234.
  • 7Zausinger S, Lumenta DB, Pruneau D, et al. Effects of LF 16- 0687 Ms, a bradykinin B2 receptor antagonist, on brain edema formation and tissue damage in a rat model of temporary focal cerebral ischemia[ J ]. Brain Res, 2002,950 ( 1 - 2) :268 - 278.
  • 8Xia CF, Yin H, Borlongan CV. Kallikrein gene transfer protects against ischemia stroke by promoting glial cell migration and inhibiting apoptosis [ J ]. Hypertension, 2004,43 (2) : 452 - 459.
  • 9Xia CF, Yin H, Yao YY, et al. Kallikrein protects against ischemia stroke by inhibiting apoptosis and inflammation and promoting angiogenesis and neurogenesis[ J]. Human Gene Therapy, 2006, 17(2) :206 -219.
  • 10Kochanek PM, Berger RP, Bayr H, et al. Biomarkers of primary and evolving damage in traumatic and ischemic brain injury: diagnosis, prognosis, probing mechanisms, and therapeutic decision making[J]. Curr Opin Crit Care, 2008, 14(2) :135 -141.

共引文献1

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部