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Enhanced neuronal degradation of amyloid-β oligomers allows synapse regeneration

Enhanced neuronal degradation of amyloid-β oligomers allows synapse regeneration
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摘要 The amyloid hypothesis of Alzheimer's disease (AD) pathogen- esis maintains that the key event is the production of specific C-terminal amyloid-β (Aβ) peptides following the abnormal proteolytic cleavage of the amyloid precursor protein (Vassar and Citron, 2000). The amyloid hypothesis of Alzheimer's disease (AD) pathogen- esis maintains that the key event is the production of specific C-terminal amyloid-β (Aβ) peptides following the abnormal proteolytic cleavage of the amyloid precursor protein (Vassar and Citron, 2000).
作者 Clive Bate
出处 《Neural Regeneration Research》 SCIE CAS CSCD 2015年第5期700-701,共2页 中国神经再生研究(英文版)
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参考文献29

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