期刊文献+

内皮素与充血性心力衰竭 被引量:1

下载PDF
导出
摘要 内皮素与充血性心力衰竭的发生发展及疾病的预后密切相关。抑制内皮素的作用可改善心衰患者的血流动力学 ,缓解患者的症状 。
出处 《心脏杂志》 CAS 2003年第3期262-264,共3页 Chinese Heart Journal
  • 相关文献

参考文献13

  • 1林丽,袁文俊.内皮素-1与心肌电活动、心肌缺血的关系[J].心脏杂志,2000,12(5):398-400. 被引量:10
  • 2Verhaar MC, Strachan FE, Newby DE, et al. Endothelin-A receptor antagonist-mediated vasodilation is attenuated by inhibition of nitric oxide synthesis and by endothelin-B receptor blockade[J]. Circulation, 1998,97(8):752--756.
  • 3Ponicke K, Vogelsang M, Heiroth M, et al. Endothelin receptors in the failing and nonfailing heart[J]. Circulation, 1998,97:744--751.
  • 4MacCarthy PY, Grocott-Mason R, Prendergast BD, et al. Contrasting inotropic effects of endogeneous endothelin in the normal and failing human heart[J]. Circulation, 2000, 101:142--147.
  • 5Kobayshi T, Miysuchi T, Sakai S, et al. Downregulation of ET(B)receptor, but not ET(A) receptor, in congetive lung secondary to heart failure. Are marked increase in circulating endothelin-1 partly attributable to decrease in lung ET(B) receptor-mediated clearance of endothelin-1?[J]. Life Sci,1998,62:185--193.
  • 6Donckier JE. Therapeutic role of bosentan in hypertension, lesions from the model of perinephritic hypertension[J]. Heart Fail Rev, 2001,6 :253-- 264.
  • 7Pernow J, Wang QD. Endothelin in myocardial ischemia and reperfusion[J]. Cardiovasc Res, 1997,33: 518.
  • 8Spiekar LE, Mitrovic V, Noll G, et al. Acute hemodynamic and neurohumoral effects ofselective ETA-receptor blockade in patients with congestive heart failure [J]. J Am Coll Cardiol,2000,35: 1745--1752.
  • 9Zolk O, Quattek J, Sitzler G, et al. Expression of endothelin-- 1, endothelin-converting enzyme, and endothlin receptors in chronic heart failure[J]. Circulation,1999,99:2118--2123.
  • 10Packer M, Caspi A, Charlon V, et al. Multicenter, doubleblind, placebo-controlled study of long-term endothelin blockade with bosentan in chronic heart failure-results of the REACH-1 trial[J]. Circulation, 1998,98 (Supp1) : S12.

二级参考文献18

  • 1YanagizawaM,KuriharaH,KimuraS,etal.Anovelpotentconstrictorpeptideproducedbyvasothelialcells[J].Nature,1988,332(6163):411.
  • 2CramerH,MullerEsterlW,SchroederC.Subtype-specificendothelin-Aandendothelin-Breceptordesensitizationcorrelateswithdifferentialreceptorphosphorylation[J].JcardiovascPharmacol,1998,31(Suppl1):S203.
  • 3KarneS,JayawickremeCK,LernerMR.Cloningandcharacterizationofanendothelin-3specificreceptor(ETcreceptor)fromxenopuslaevisdermalmelanophores[J].JbiolChem,1993,268(25):19126,
  • 4Ruiz-OpazoN,HirayamaK,AkimotoK,etal.Molecularcharacterizationofadualendothelin-1/angiotensinⅡreceptor[J].MolMed,1998,4(2):96.
  • 5NakajimaT,HazamaH,HamadaE,etal.Endothelin-1andvasopressinactivateCa2+-permeablenon-selectivecationchannelsinaorticsmoothmusclecells:mechanismofreceptor-mediatedCa2+influx[J].JMolCellCardiol,1996,28(4):707.
  • 6KobayahiS,NakayaH,TakizawaT,etal.Endothelin-1partiallyinhibitsATP-sensitiveK+currentinguineapigventricularcells[J].JCardiovascPharmacol,1996,27(1):12.
  • 7WatanukiM,HorieM,TsuchiyaK,etal.Endothelin-1inhibitionofcardiacATP-sensitiveK+channelsviapertussis-toxin-sensitiveG-proteins[J].CardiovascRes,1997,33:123.
  • 8YorikaneR,ShigaH,MiyakeS,etal.Evidencefordirectarrhythmogenicactionofendothelin[J].BiochemBiophysResCommun,1990,173(1):457.
  • 9OnoK,TsujimotoG,SakamotoA,etal.Endothelin-Areceptormediatescardiacinhibitionbyregulatingcalciumandpotassiumcurrents[J].Nature,1994,370(6487):301.
  • 10JamesA,XieLH,FujitaneY,etal.InhibitionofthecardiacproteinkinaseA-dependentchlorideconductancebyendothelin-1.Nautre,1994,370(6487):297.

共引文献9

同被引文献9

  • 1Milano G, Corno AF, Lippa S, et al. Chronic and intermittent hypoxia induce different degrees of myocardial tolerance to hypoxia-induced dysfunction [ J ]. Exp Biol Med, 2002,227(6):389-397.
  • 2Ostadal B, Ostadalova I, Dhalla NS. Development of cardiac sensitivity to oxygen deficiency: comparative and ontogenetic aspects [ J ].Physiol Rev, 1999, 79(3):635 - 659.
  • 3Baker JE, Hohnan P, Kalyanaraman B, et al. Adaptation to chronic hypoxia confers tolerance to subsequent myocardial ischemia by increased nitric oxide production [J]. Ann N Y Acad Sci, 1999,874:236 - 253.
  • 4Giaid A. Nitric oxide and endothelin-1 in puhnonaty hypertension[J]. Chest,1998,114: 208S - 212S.
  • 5Zhu WD, Zou YZ, Shiojima I, et al. Ca^2+ cahnoduhn-dependent kinase II and calcineurin play critical roles in endothelin-1-induced cardiomyocyte hypertrophy [ J ]. J Biol Chem, 2000, 275( 20 ):15239 - 15245.
  • 6Delgado C, Artiles A, Gomez AM, et al. Frequency-dependent increase in cardiac Ca^2+ current is clue to reduced Ca^2+ release by the sarcoplasmic reticulum[J]. J Mol Cell Cardiol, 1999,31(10):1783-1793.
  • 7Huhne JT, Orchard CH. Effect of acidosis on Ca^2+ uptake and release by sarcoplasmic reticulum of intact rat ventricular myocytes[ J ]. Am J Pbysiol, 1998,275(3 Pt 2): H977 - H978.
  • 8Sica AL, Greenberg HE, Ruggiero DA, et al. Chronic - intermittent hypoxia: a model of sympathetic activation in the rat[J]. Res Physiol, 2000,121: 173 - 184.
  • 9Prabhakar NR, Fields RD, Baker T, et al. Intermittent hypoxia: cell to system[J]. Am J Physiol Lung Cell Mol Physiol,2001,281: L524-L528.

引证文献1

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部