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进展性卒中 被引量:8

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作者 苏志强
出处 《黑龙江医学》 2004年第2期89-91,共3页 Heilongjiang Medical Journal
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参考文献8

  • 1[1]Birschel P, Ellul J, Barer D. Progressing Stroke: Towards an Internationally Agreed Definition[J]. Cerebrovasc Dis, 2004,17(2~3):242~252.
  • 2[2]Audebert H J, Pellkofer T S, Wimmer M L,et al.Progression in Lacunar Stroke Is Related to Elevated Acute Phase Parameters[J]. Eur Neurol, 2004,51(3):125~131.
  • 3[3]Matsumoto N, Kimura K, Yokota C, et al. Early neurological deterioration represents recurrent attack in acute small non-lacunar stroke[J]. J Neurol Sci, 2004,15:217(2):151~5.
  • 4[4]Sumer M, Ozdemir I, Erturk O. Progression in acute ischemic stroke: Frequency, risk factors and prognosis[J].J Clin Neurosci. 2003,10(2):177~80.
  • 5[5]Barber M, Wright F, Stott D J, et al. edictors of early neurological deterioration after ischaemic stroke: a case-control study[J]. Gerontology, 2004, 50(2):102~9.
  • 6[6]Caplan L R. Worsening in ischemic stroke patients: is it time for a new strategy?[J]. Stroke, 2002,33(6):1 443~5.
  • 7徐静红,陈淑慧,许玉杰.进展性脑卒中与肺感染的关系[J].中国煤炭工业医学杂志,2001,4(7):519-519. 被引量:15
  • 8王洪新,韩仲岩.高血压、糖尿病与进展性脑卒中的相关研究[J].卒中与神经疾病,2002,9(6):364-365. 被引量:64

二级参考文献13

  • 1脑卒中患者临床神经功能缺损程度评分标准(1995)[J].中华神经科杂志,1996,29(6):381-383. 被引量:15752
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  • 4Carberg B, Asplund K, Hagge E. Factors influencing admission blood pressure levels in acute stroke. Stroke, 1991, 22(4):527
  • 5Philips SJ. Pathophysiology and management of hypertension in acute ischemic stroke. Hypertension, 1994, 23 ( 1 ): 131
  • 6Ceriello A, Falleti E, Bortolotti N, et al. Increased circulating intercellular adhesion molecule-lexpression in cultured human vascular endothelial cells. Cells J clin Invest, 1994, 98:885
  • 7Mcleod Ds, Lefer DJ, Merges C, et al. Enhanced expression of ihtercellular adhesion molecule-land P-selectin in the diabetic human retina and chroid. AM J Pathol, 1995, 147:642
  • 8Linn T, Stmate C, Federlin K, et al. Intercellular adhesion molecule-1, expression in the islets of nonobese diabetes and lowdosestretozocin-treated mouse. Histochemistry, 1994, 102:317
  • 9Herold K, Vezys V, Gage A, et al. Prevention of autoimmune diabetes by treatment with anti-LFA-1 and anti-ICAM-1 monoclonal antibody. Cell Immunoi, 1994, 157:489
  • 10Matsui H, Suzuki M, Tsukuda R, ET al. expression of ICAM-1 on glumeruli isassociated with pyogression of diabetic nephropathy in a genetically obese diabetic rats wister fatty. Diabetes Res clin pract,1996, 32(1-2):1

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二级引证文献89

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