Past monoculture forestry in China has contributed to countrywide ecological disasters and economic difficulties in forestry regions. China's new forestry programs, Natural Forest Conservation Program and Returnin...Past monoculture forestry in China has contributed to countrywide ecological disasters and economic difficulties in forestry regions. China's new forestry programs, Natural Forest Conservation Program and Returning Farmlands to Forests Program, provide opportunities for ecosystem management of mountain forests in China. A decision support system, FORE-STAR, has been developed for better managing and protecting natural forests in Changbai Mountain area. It uses GIS-based forest inventory data at a scale of forestry bureau. The first version contains two sub-modules: forest operation and forest res-toration. Under each sub-module, users can compare several decision options and make optimal choices. It can help field for-esters, forest managers, and policy makers make multi-objective and consistent decisions in planning forest management at hierarchical administrative scales.展开更多
The experiments were conducted to assess the influences of thyroid hormone on cardiac protein kinase C(PKC) signal pathway with cultured cardiac myocytes and fibroblasts as the models. Cells were pretreated with 1% ne...The experiments were conducted to assess the influences of thyroid hormone on cardiac protein kinase C(PKC) signal pathway with cultured cardiac myocytes and fibroblasts as the models. Cells were pretreated with 1% newborn calf serum (NCS) or angiotensin II (Ang II), and then following by a triiodothyronine (T3) treatment. The PKC activity, PKCα and PKCε expressions were analyzed and compared. In 1% NCS pretreatment, T3 could inhibit PKC activity and PKCε expression in cardiac myocytes. The AngII pretreatment led to an increase of PKC activity and PKCε expression in cardiac myocytes, and an increase of PKC activity in cardiac fibroblasts. Following by T3 treatment, the increased PKC activity and PKCε expression in cardiac myocytes were markedly decreased. In conclusion, whether in 1% NCS or in Ang II pretreatment, T3 could inhibit PKC activity and PKCε expression in cardiac myocytes. Key words thyroid hormone - cardiac myocytes - cardiac fibroblasts - protein kinase C CLC number Q 572 Foundation item: Supported by the Natural Science Foundation of Hubei Province (98091)Biography: WANG Bao-hua (1974-), female, Ph. D, research direction: cardiovascular pathophysiology.展开更多
文摘Past monoculture forestry in China has contributed to countrywide ecological disasters and economic difficulties in forestry regions. China's new forestry programs, Natural Forest Conservation Program and Returning Farmlands to Forests Program, provide opportunities for ecosystem management of mountain forests in China. A decision support system, FORE-STAR, has been developed for better managing and protecting natural forests in Changbai Mountain area. It uses GIS-based forest inventory data at a scale of forestry bureau. The first version contains two sub-modules: forest operation and forest res-toration. Under each sub-module, users can compare several decision options and make optimal choices. It can help field for-esters, forest managers, and policy makers make multi-objective and consistent decisions in planning forest management at hierarchical administrative scales.
文摘The experiments were conducted to assess the influences of thyroid hormone on cardiac protein kinase C(PKC) signal pathway with cultured cardiac myocytes and fibroblasts as the models. Cells were pretreated with 1% newborn calf serum (NCS) or angiotensin II (Ang II), and then following by a triiodothyronine (T3) treatment. The PKC activity, PKCα and PKCε expressions were analyzed and compared. In 1% NCS pretreatment, T3 could inhibit PKC activity and PKCε expression in cardiac myocytes. The AngII pretreatment led to an increase of PKC activity and PKCε expression in cardiac myocytes, and an increase of PKC activity in cardiac fibroblasts. Following by T3 treatment, the increased PKC activity and PKCε expression in cardiac myocytes were markedly decreased. In conclusion, whether in 1% NCS or in Ang II pretreatment, T3 could inhibit PKC activity and PKCε expression in cardiac myocytes. Key words thyroid hormone - cardiac myocytes - cardiac fibroblasts - protein kinase C CLC number Q 572 Foundation item: Supported by the Natural Science Foundation of Hubei Province (98091)Biography: WANG Bao-hua (1974-), female, Ph. D, research direction: cardiovascular pathophysiology.
