来茂德教授和张红河副教授在《胃肠道》(Gut)在线发表了题为“SRSF6-Regulated Alternative Splicing that Promotes Tumor Progression Offers a Therapy Target for Colorectal Cancer'(http://gut.bmj.com/content/early/2017/11...来茂德教授和张红河副教授在《胃肠道》(Gut)在线发表了题为“SRSF6-Regulated Alternative Splicing that Promotes Tumor Progression Offers a Therapy Target for Colorectal Cancer'(http://gut.bmj.com/content/early/2017/11/07/gutjnl-2017-314983)的研究论文,揭示了SRSF6作为结直肠癌治疗靶点调控RNA可变剪接的分子机制。该研究在前期研究IncRNA LINC01133通过结合可变剪接因子SRSF6抑制结直肠癌内皮间质转化和转移(Kong J,Sun W,Li C,et al.Long non-coding RNA LINC01133 inhibits epithelial-mesenchymal transition and metastasis in展开更多
文摘来茂德教授和张红河副教授在《胃肠道》(Gut)在线发表了题为“SRSF6-Regulated Alternative Splicing that Promotes Tumor Progression Offers a Therapy Target for Colorectal Cancer'(http://gut.bmj.com/content/early/2017/11/07/gutjnl-2017-314983)的研究论文,揭示了SRSF6作为结直肠癌治疗靶点调控RNA可变剪接的分子机制。该研究在前期研究IncRNA LINC01133通过结合可变剪接因子SRSF6抑制结直肠癌内皮间质转化和转移(Kong J,Sun W,Li C,et al.Long non-coding RNA LINC01133 inhibits epithelial-mesenchymal transition and metastasis in
